Alcohol Withdrawal and Brain Injuries: Beyond Classical Mechanisms
Description:
This review attempts to provide integrated information at the levels of oxidative signaling mechanisms by which ethanol withdrawal (EW) provokes brain injuries and 17β-estradiol (E2) protects against it. Unmanaged sudden withdrawal from the excessive consumption of alcohol (ethanol) adversely alters neuronal integrity in vulnerable brain regions such as the cerebellum, hippocampus, or cortex.
Date:
July 20, 2010
Creator:
Jung, Marianna E. & Metzger, Daniel B.
Item Type:
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Partner:
UNT Health Science Center