Report on the Agricultural Experiment Stations, 1953 Page: 18
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18 REPORT ON EXPERIMENT STATIONS, 1953
by the Indiana station, is probably caused by the great length of the
sex chromosome. The definite linkage between Shaker and latefeathering
supports evidence for the belief that separate genes at
different loci produce the Jittery and Shaker traits. These accumulating
facts make it possible for the pedigree poultry breeder to eliminate
lethal genes from his breeding flock by progeny testing.
Nutrition and Disease Control
Practically all of the State experiment stations are engaged in phases
of research dealing with the interrelation between poultry nutrition
and health. A few typical examples follow.
Ascites in turkeys
Eight experiments have been conducted by the Pennsylvania station
to learn more about the relation between nutrition and ascites (dropsy)
in turkeys. The relative toxicity of seven salts fed singly or in combinations
was tested. The sodium ion was found to be the agent responsible.
It was more toxic in the form of chloride salt than in the
form of either citrate or acetate salt.
The Louisiana station undertook an experiment on the effects of
two different levels of nutrient intake on the performance of laying
hens. The flocks fed a low level of nutrient intake, approximately 75
percent of the allowance recommended by the National Research
Council (NRC) for all nutrients except energy, had twice the number
of deaths from "pullet disease" or "blue comb" as the flock receiving
a high level of intake that contained 125 percent of the NRC recommended
Articular and cervical paralysis in geese
Goslings fed the standard chick-starting ration adopted by the
Minnesota station frequently develop hock disorder when brooded
either in batteries or on litter. Post-mortem examination revealed,
in each case, that the tendon of the tibia had slipped out of the
condyle, justifying the application of the term "perosis," to the condition.
In research designed to determine the cause of this malady,
84.6 percent of all goslings fed the basal diet alone developed perosis
in 3 weeks. The addition of 40 milligrams of niacin (nicotinic acid)
per kilogram of diet completely prevented the perotic condition.
The Minnesota station also found that a deficiency of either choline
or pteroylglutamic acid (folic acid) will cause goslings to develop
an articular paralysis due chiefly to involvement of the hock joint.
Goslings on a pteroylglutamic acid deficient diet, in addition to having
weak legs, also manifested a weakness of the neck. This type
of paralysis of the neck was cured by an intramuscular injection of
crystalline pteroylglutamic acid.
Hock disorder in turkeys
The Washington station has found that supplementation of the diet
of turkeys with 75 milligrams of niacin and 50 milligrams of vitamin
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United States. Office of Experiment Stations. Report on the Agricultural Experiment Stations, 1953, book, 1953; Washington D.C.. (digital.library.unt.edu/ark:/67531/metadc5989/m1/20/: accessed August 21, 2017), University of North Texas Libraries, Digital Library, digital.library.unt.edu; crediting UNT Libraries Government Documents Department.