Serotonin, Norepinephrine, and the Hypothalamic Ventromedial Nucleus: a Proposed Mechanism Mediating Hyperphagia and Obesity Page: 41
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41
periventricularis nucleus, and sometimes extended so far
laterally that it hit parts of the hypothalamic lateral
nucleus. Sometimes, the lesion tracks were large and
obliterated quite a large pathway. The lesions averaged an
anterior-posterior distance of 2000 microns. Because of
differences in lesions, data for the two runs were analyzed
separately.
Diet
Statistics
A separate three-way analysis of variance (ANOVA) was
performed for each run on the independent variables DIET,
LESION (lesioned and unlesioned conditions), and DAY, for
the dependent variables water intake, food intake, and gross
animal weight. (See Figure 7, pg 43). DAY was a repeated
measure for each of the three dependent variables, and
LESION and DIET were random factors. Significant
differences were found for LESION and DIET. For this reason
the three-way ANOVA was broken up into two two-way ANOVAs.
First, LESION was compared for each dependent variable over
the fourteen day period for each diet. (Figure 7B,pg 43).
Second, DIET was compared for each dependent variable over
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McDermott, Kathy Howard. Serotonin, Norepinephrine, and the Hypothalamic Ventromedial Nucleus: a Proposed Mechanism Mediating Hyperphagia and Obesity, thesis, May 1981; Denton, Texas. (https://digital.library.unt.edu/ark:/67531/metadc504188/m1/47/: accessed April 25, 2024), University of North Texas Libraries, UNT Digital Library, https://digital.library.unt.edu; .