Chronic Excess Salt Consumption as an Etiologic Factor in Human Hypertension

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Description

Since 1940 it has been known that extra salt (i.e. sodium chloride) facilitated the development of experimental hypertension induced by various sterols; subsequently hypertension was induced by desoxycorticosterone acetate plus sodium chloride; then hypertension was produced by using hypertonic saline as the sole source of liquid; and finally, in 1953, Menesly and his associates reported that chronic ingestion of excess dietary salt alone would produce hypertension in rats. Furthermore, in conformity with general pharmacologic experience relating dose response to successive increments of a drug, as the average daily salt intake increased, the average systolic, blood pressure increased.

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Dahl, Lewis K. February 1, 1963.

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Since 1940 it has been known that extra salt (i.e. sodium chloride) facilitated the development of experimental hypertension induced by various sterols; subsequently hypertension was induced by desoxycorticosterone acetate plus sodium chloride; then hypertension was produced by using hypertonic saline as the sole source of liquid; and finally, in 1953, Menesly and his associates reported that chronic ingestion of excess dietary salt alone would produce hypertension in rats. Furthermore, in conformity with general pharmacologic experience relating dose response to successive increments of a drug, as the average daily salt intake increased, the average systolic, blood pressure increased.

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  • February 1, 1963

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  • Feb. 5, 2022, 9:41 a.m.

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  • Sept. 25, 2022, 3:52 a.m.

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Dahl, Lewis K. Chronic Excess Salt Consumption as an Etiologic Factor in Human Hypertension, report, February 1, 1963; Washington D.C.. (https://digital.library.unt.edu/ark:/67531/metadc1250506/: accessed April 18, 2025), University of North Texas Libraries, UNT Digital Library, https://digital.library.unt.edu; crediting UNT Libraries Government Documents Department.

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